(ISSN 2573-4407)
OBM Neurobiology is an international peer-reviewed Open Access journal published quarterly online by LIDSEN Publishing Inc. By design, the scope of OBM Neurobiology is broad, so as to reflect the multidisciplinary nature of the field of Neurobiology that interfaces biology with the fundamental and clinical neurosciences. As such, OBM Neurobiology embraces rigorous multidisciplinary investigations into the form and function of neurons and glia that make up the nervous system, either individually or in ensemble, in health or disease. OBM Neurobiology welcomes original contributions that employ a combination of molecular, cellular, systems and behavioral approaches to report novel neuroanatomical, neuropharmacological, neurophysiological and neurobehavioral findings related to the following aspects of the nervous system: Signal Transduction and Neurotransmission; Neural Circuits and Systems Neurobiology; Nervous System Development and Aging; Neurobiology of Nervous System Diseases (e.g., Developmental Brain Disorders; Neurodegenerative Disorders).
OBM Neurobiology publishes a variety of article types (Original Research, Review, Communication, Opinion, Comment, Conference Report, Technical Note, Book Review, etc.). Although the OBM Neurobiology Editorial Board encourages authors to be succinct, there is no restriction on the length of the papers. Authors should present their results in as much detail as possible, as reviewers are encouraged to emphasize scientific rigor and reproducibility.
Publication Speed (median values for papers published in 2024): Submission to First Decision: 7.6 weeks; Submission to Acceptance: 13.6 weeks; Acceptance to Publication: 6 days (1-2 days of FREE language polishing included)
Special Issue
Neuroimmunology and Neuroinflammation: Mechanisms and Therapeutic Advances
Submission Deadline: August 31, 2026 (Open) Submit Now
Guest Editor
Vittorio Gentile, MD, PhD
Departments of Precision Medicine, CIRN - Interuniversity Center of Neuroscience Researches, University of Campania "Luigi Vanvitelli", via Luigi De Crecchio 7, 80138 Naples, Italy
Research Interests: Transglutaminases; neurodegenerative diseases; neurobiology
About This Topic
Neuroinflammation seems to play a pivotal role in various chronic neurodegenerative diseases, characterized also by the pathogenetic accumulation of specific protein aggregates. Several of these proteins have been shown to be substrates of transglutaminases, calcium-dependent enzymes that catalyze protein crosslinking reactions. Recently, it has been demonstrated that Transglutaminase 2 (TG2), a member of the transglutaminase enzymes family, may also be involved in molecular mechanisms underlying inflammation. In the central nervous system, microglia and astrocytes are the cell types mainly involved in the inflammatory process. The suggested topic is focused on the increased expression of inflammatory genes, such as TG2 increased expression and enzyme activity, that occur in neuronal and non-neuronal cells, such as glial, microglial and monocyte cells, in response to inflammatory stimuli. Moreover, we will focus on the discovery and analysis of neuroprotective effects of natural compounds and nutraceuticals against toxic insults and their anti-inflammatory activities, through the modulation of genes responsible for inflammation and oxidative stress.
Keywords
Neuroinflammation; neurodegeneration; nutraceuticals
Manuscript Submission Information
Manuscripts should be submitted through the LIDSEN Submission System. Detailed information on manuscript preparation and submission is available in the Instructions for Authors. All submitted articles will be thoroughly refereed through a single-blind peer-review process and will be processed following the Editorial Process and Quality Control policy. Upon acceptance, the article will be immediately published in a regular issue of the journal and will be listed together on the special issue website, with a label that the article belongs to the Special Issue. LIDSEN distributes articles under the Creative Commons Attribution (CC BY 4.0) License in an open-access model. The authors own the copyright to the article, and the article can be free to access, distribute, and reuse provided that the original work is correctly cited.
Submitted manuscripts should not have been published previously, nor be under consideration for publication elsewhere (except conference proceedings papers). Research articles and review articles are highly invited. Authors are encouraged to send the tentative title and abstract of the planned paper to the Editorial Office (neurobiology@lidsen.com) for record. If you have any questions, please do not hesitate to contact the Editorial Office.
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2023 
CiteScore SJR SNIP 1.0 0.232 0.256 - Newsletter